Our research program is focused on the immunological processes and molecular mechanisms underlying lung repair and resolution following acute lung injury.

Major areas of interests include:

• Studies investigating the mechanisms of Foxp3⁺ regulatory T cells (Tregs) in promoting resolution of lung injury
• Lymphocyte interactions with the distal lung epithelium
• Clinical and translational studies of Acute Respiratory Distress Syndrome (ARDS)

Identifying and elucidating mechanisms of lung resolution will allow for the potential development of therapeutic approaches to minimize collateral tissue damage without adversely altering the beneficial response to injury.